bothered to look for the study taking the article at "face value."
The study cites some very disturbing evidence of other issues with Advanced Paternal Age (APA) that isn't even touched in the original article.
I also find it entertaining to read the posters describing themselves as intelligent offspring of older fathers yet who didn't bother to spend the approximately 2 minutes it took to google the study and only replied "Nuh, uh! I'm brilliant as are my siblings!" If brilliant is defined as intellectually incurious and lazy mixed with a willingness to accept pablum packaged as "journalism," I guess they fit their definition.
The second paragraph in this snippet is most disturbing.
In recent decades there has been increased attention to health outcomes in the offspring of older fathers. Evidence shows that advanced paternal age (APA) is associated with an increased risk of a wide range of disorders <1>. While not discounting the influence of various age-related psychosocial factors that may translate to differential health outcomes for the offspring of older fathers (e.g., higher socioeconomic status, better education), advances in genomics have refocused attention on the vulnerability of sperm from older fathers to carrying de novo mutations. The development of the germ cell differs between human males and females—there are many more germline cell divisions in the life history of a sperm relative to that of an oocyte <2>. In the female there are 22 mitotic cell divisions that occur in utero. In contrast, after puberty, progenitor sperm stem cells undergo mitotic cell division once every 16 d. By age 20 the progenitor sperm cells have undergone approximately 150 cell divisions. By age 50 this number is 840. Thus, the chance of copy error mutations increases with age in males more dramatically than for females.
Advanced paternal age is associated with increased fetal deaths <3,4> and certain rare congenital syndromes (e.g., achondroplasia) <1,5>. In recent years evidence has accumulated linking APA with a wide range of neurological and neuropsychiatric conditions including Alzheimer's disease <6,7>, bipolar disorder <8>, dyslexia <9>, neural tube defects <10>, and epilepsy <11>. A sizeable body of evidence has accumulated linking APA with an increased risk of schizophrenia <12–18>. A recent meta-analysis based on eight studies found that paternal age above 35 was associated with an increased risk of schizophrenia <19>. There is also evidence linking APA to autism spectrum disorders <20–24>.
The associations between APA and outcomes such as autism and schizophrenia are of particular interest, as these disorders have recently been associated with genomic structural variation <25–30>. It is feasible that APA-related mechanisms may contribute to genomic structural variation (e.g., copy number variants, microdeletions) <2>. Thus, within the fields of schizophrenia and autism research, there has been an unexpected convergence between epidemiology and molecular biology.
While there is good evidence linking paternal age with several clinically distinct neurodevelopmental disorders, the evidence linking paternal age and other neurocognitive outcomes such as general intelligence is less robust. Earlier studies noted an association between APA and poorer performance on neurocognitive tests <31–34>. This issue has been addressed specifically in a recent study based on male and female Israeli conscripts (age 16–17 y, n = 44,175) <35>. The study found independent effects of paternal age on offspring intelligence with the lowest scores associated with both younger and older fathers (inverted “U”-shaped association). This finding is in contrast to the association between maternal age and offspring intelligence, where most studies have reported a linear association between older maternal age and superior neurocognitive ability <36–39>.
Link to the study that is actually online